Fig 1: IFN-? effects on the protein levels of IL-1ß, IL-6, IL-8 and IL-18. C. trachomatis-infected synovial cells pretreated with IFN-? as well as untreated or uninfected cells were assayed via ELISA. * p < 0.05, ** p < 0.001, *** p < 0.0001 and **** p < 0.000001 vs. C. trachomatis infected cells.
Fig 2: IFN-? effects on the relative mRNA expression of IL-1ß, IL-18, IL-6, IL-8 and caspase-1. C. trachomatis infected synovial cells pretreated with IFN-? as well as untreated or uninfected cells were assayed via reverse transcription real-time PCR. * p < 0.05, ** p < 0.01, *** p < 0.0001 and **** p < 0.00001 vs. C. trachomatis infected cells.
Fig 3: Expression of inflammatory factors in patients during the perioperative period. (a) Expression of IL-1ß. (b) Expression of IL-6. (c) Expression of TNF-a. (d) Expression of IL-10. Comparison between the two groups, *P < 0.05.
Fig 4: Relative mRNA expression of IL-1ß, IL-18, IL-6, IL-8 and caspase-1 following C. trachomatis infection of synovial cells. Results are expressed as fold changes, as compared to uninfected cells, of mRNA expression levels at 6, 18 and 24 h.p.i., via reverse transcription real-time PCR. *, p < 0.05; **, p < 0.01.
Fig 5: Effects of CAE on the secretion of (a) interleukin (IL)-1ß and (b) IL-6 in IFN-?- and TNF-a-induced human skin keratinocytes after 24 h. ##, p < 0.01 vs. non-induced group. *, p < 0.05 vs. IFN-?/TNF-a-induced group. CAE decreased the IFN-?- and TNF-a-induced IL-1ß and IL-6 levels in HaCaT cells.
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